Autoimmune Disease

Natural  and  normal  functions  of  immune  system depend on self-tolerance, and self-tolerance deficiency can  result  in  autoimmunity.  Autoimmune  disease  studies  in  both  monozygotic  and dizygotic  twins  have suggested  a  role for  epigenetic factors. Epigenetic homeostasis  failure, as a  response to  environmental agents, can result in gene expression changes  in  specific  differentiated  cells  leading  to dysregulated self-tolerance.  The immune system and  target organ are two  main players  in  an  autoimmune  disease  process  and  the epigenetic  modifications  of these  players could  have roles  in  disease  development.  Many  functions  of immune  cells  such  as  hematopoietic  lineage, rearrangement  of  antigen-receptor,  allelic  exclusion, and inducible immune responses against pathogens are epigenetically controlled. The alterations of epigenetic mechanisms  regulating  immunological  development could promote autoimmunity disease.


 



 


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